Nursing care of a child with alteration on oxygenation related to cardiac anomalies (Cyanotic)
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Occurs when blood is shunted from the venous to the arterial system as a result of abnormal communication bet. the two systems (deoxy to oxy. R to L shunts)
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Decreased pulmonary blood flow (Right to left shunt)
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Localized narrowing of the aorta, the most common cardiac malformation responsible for cyanosis in a child over 1 year. 4 major defects:
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Pulmonary stenosis
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Ventricular septal defect
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Aorta overriding VSD
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Right ventricular hypertrophy
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Pathophysiology
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•PS impedes the flow of blood to lungs
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•increases pressure in the right ventricle (hypertrophy)
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•Forces deoxy blood through the septal defect to the left ventricle
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•Overriding aorta receives blood from both right and left ventricles.
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Signs & Symptoms
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•Clubbing of the fingers, murmur
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•Severe dyspnea, hypercyanotic spells & acidosis
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•Polycythemia & clot formation
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•Failure to thrive and growth retardation.
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Treatments
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•If O2 level ex low, prostaglandins (IV) to keep PDA open.
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•Complete repair when 6 mos. of age.
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•Closure of the VSD with dacron patch
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•The narrowed pulmonary valve is enlarged
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•Coronary arteries will be repaired
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•Hypertrophy of right heart should remodel when pressure in right side is reduced.
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Streptococcal infections causes damage to the heart muscle and valves. All heart layers are affected.
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•Pancarditis (all layers)
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•Pericarditis
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•Myocarditis
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•Endocarditis
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Pathogenesis
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•An acute attack of streptococcal pharyngitis by group A beta-hemolytic streptococci
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•Within 2-4 weeks after this attack anti-streptococcal antibodies are formed and attack the heart and the extra cardiac sites.
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Diagnostic test
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•Throat swab, blood test, ECG heart test
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Tricuspid valve fails to develop causing complete closure of the tricuspid valve that results in mixed blood flow.
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Signs & Symptoms
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•Cyanosis in NB, Chronic hypoxia, clubbing
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•No blood flow form the right A to the right V
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•Heart failure, chronic hypoxemia
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•Failure to thrive and growth retardation.
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Diagnosis
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•Listening with a steth for changes in heart sounds
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•O2 sat to see how much O2 is getting into blood
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•Chest x-ray to see the size & position of the heart
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•ECG to check the electrical activity
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•Ultrasound scan (echocardiography)
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Treatment
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•A glenn | Cavopulmonary | HemiFontan shunt
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making a passage between the superior vena cava to the right lung (pulmonary) artery
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•A fontan | Total Cavo-Pulmonary Connection
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This operation joins the inferior vena cava to the pulmonary arteries.
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Nonpruritic, painless erythematous rash on trunk and/or proximal extremities.
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•Macular lesions with raised margins & central clearing
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•May last from weeks to months
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Minor Manifestations
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Clinical: Fever, arthralgia, history of RF
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Lab test: Acute phase reactants, ECG changes
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Major Manifestations
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• Carditis: friction rub, murmur, cardiomegaly, CHF
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•Arthritis: migratory polyarthritis, swollen, red, tender
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•Chorea, subcutaneous nodules, Erythema marginatum
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A neurologic disorder with muscular weakness, emotional lability and involuntary, uncoordinated, purposeless movement
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•Disappear during sleep, lasts 2-4 months
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•Mainly occurs in hands, feet and face, sensation intact
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• An autoimmune disease affecting the heart and extra cardiac sites ( joints, brain, skin)
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•Occurs 10 days to 6 weeks after pharyngitis
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•The disease affects children and young adults (5-15yrs)
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•The disease follows upper respi infection (tonsillitis)
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Cardiac Manifestations
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•Pericardial friction rubs, weak heart sounds
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•Tachycardia, arrythmias
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•Extracardiac: fever, polyarthritis, arthralgia, skin lesions, chorea
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•Pharyngeal culture may be negative
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• Anti streptolysin O (ASO) titer will be high
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•May be migratory. last for 2 - 3 weeks
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•Warm, swollen, tender joints, inflamed membrane
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•Usually involves the knees, ankles, elbows, and wrists
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•Eroded cartilage and narrowed joint space
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Usually associated with severe carditis and occur several weeks after
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•Firm, painless nodules (up to 2cm) found over bony surfaces and tendons. persist for 1-2 weeks.
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•Occur near elbows, knees, wrists, achilles tendon, vertebral joints
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Idiopathic multisystem disease. vasculitis of small & medium blood vessels & arteries. Usually <5 yrs old.
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Mucocutaneous Ocular Syndrome (MCOS) the original name of kawasaki disease. The cause is unknown.
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Diagnostic Criteria
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•Changes in extremities: edema, erythema, desquam.
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•Polymorphous exanthem, usually truncal
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•Conjunctival injection, fissuring of lips & oral cavity
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•Cervical lymphadenopathy
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Symptoms
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•Red eyes, changes in lips, tongue & mouth
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•Redness, cracking on lips, red strawberry tongue
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•Hands & feet: red, swollen, hands & soles.
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•Body rash : 1st appears w/fever. bumpy to touch
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•Swelling of glands in the neck, soft, painless nodes
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Phases
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Acute: 1-2 weeks from onset, febrile, irritable, oral changes, rash, edema/erythema of feet
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•Subacute: 2-8 weeks, desquamation, persistent arthritis or arthralgias, gradual improvement.
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• Convalescent (months to years later)
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Treatment
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•No single medicine can treatment or cure
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• Meds : Aspirin and gamma globulin
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